RT Journal Article SR Electronic T1 Host autophagosomes are diverted to a plant-pathogen interface JF bioRxiv FD Cold Spring Harbor Laboratory SP 102996 DO 10.1101/102996 A1 Yasin F Dagdas A1 Pooja Pandey A1 Nattapong Sanguankiattichai A1 Yasin Tumtas A1 Khaoula Belhaj A1 Cian Duggan A1 Maria E Segretin A1 Sophien Kamoun A1 Tolga O Bozkurt YR 2017 UL http://biorxiv.org/content/early/2017/01/30/102996.abstract AB Filamentous plant pathogens and symbionts invade their host cells but remain enveloped by host-derived membranes. The mechanisms underlying the biogenesis and functions of these host-microbe interfaces are poorly understood. Recently, we showed that PexRD54, an effector from the Irish potato famine pathogen Phytophthora infestans, binds host protein ATG8CL to stimulate autophagosome formation and deplete the selective autophagy receptor Joka2 from ATG8CL complexes. Here, we show that during P. infestans infection, ATG8CL autophagosomes are diverted to the pathogen interface. Our findings are consistent with the view that the pathogen coopts host selective autophagy for its own benefit.