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Leveraging the resolution of RNA-Seq markedly increases the number of causal eQTLs and candidate genes in human autoimmune disease: Mapping eQTLs in autoimmune disease using RNA-Seq
View ORCID ProfileChristopher A. Odhams, Deborah S. Cunninghame Graham, Timothy J. Vyse
doi: https://doi.org/10.1101/128728
Christopher A. Odhams
1Department of Medical & Molecular Genetics, King’s College London, London, UK
Deborah S. Cunninghame Graham
1Department of Medical & Molecular Genetics, King’s College London, London, UK
2Academic Department of Rheumatology, Division of Immunology, Infection and Inflammatory Disease, King’s College London, London, UK
Timothy J. Vyse
1Department of Medical & Molecular Genetics, King’s College London, London, UK
2Academic Department of Rheumatology, Division of Immunology, Infection and Inflammatory Disease, King’s College London, London, UK
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Posted April 19, 2017.
Leveraging the resolution of RNA-Seq markedly increases the number of causal eQTLs and candidate genes in human autoimmune disease: Mapping eQTLs in autoimmune disease using RNA-Seq
Christopher A. Odhams, Deborah S. Cunninghame Graham, Timothy J. Vyse
bioRxiv 128728; doi: https://doi.org/10.1101/128728
Leveraging the resolution of RNA-Seq markedly increases the number of causal eQTLs and candidate genes in human autoimmune disease: Mapping eQTLs in autoimmune disease using RNA-Seq
Christopher A. Odhams, Deborah S. Cunninghame Graham, Timothy J. Vyse
bioRxiv 128728; doi: https://doi.org/10.1101/128728
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