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Functionally redundant control of cardiac hypertrophic signaling by inositol 1,4,5-trisphosphate receptors
M. Iveth Garcia, Anja Karlstaedt, View ORCID ProfileJavier Amione-Guerra, View ORCID ProfileKeith A. Youker, View ORCID ProfileHeinrich Taegtmeyer, View ORCID ProfileDarren Boehning
doi: https://doi.org/10.1101/075044
M. Iveth Garcia
aCell Biology Graduate Program, University of Texas Medical Branch, Galveston, TX 77555;
bDepartment of Biochemistry and Molecular Biology, McGovern Medical School at UTHealth, Houston, TX 77030;
Anja Karlstaedt
cDepartment of Internal Medicine, Division of Cardiology, McGovern Medical School at UTHealth, Houston, TX 77030.
Javier Amione-Guerra
dHouston Methodist Hospital, Houston, TX 77030.
Keith A. Youker
dHouston Methodist Hospital, Houston, TX 77030.
Heinrich Taegtmeyer
cDepartment of Internal Medicine, Division of Cardiology, McGovern Medical School at UTHealth, Houston, TX 77030.
Darren Boehning
bDepartment of Biochemistry and Molecular Biology, McGovern Medical School at UTHealth, Houston, TX 77030;
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Posted September 13, 2016.
Functionally redundant control of cardiac hypertrophic signaling by inositol 1,4,5-trisphosphate receptors
M. Iveth Garcia, Anja Karlstaedt, Javier Amione-Guerra, Keith A. Youker, Heinrich Taegtmeyer, Darren Boehning
bioRxiv 075044; doi: https://doi.org/10.1101/075044
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