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Beyond the myosin mesa: a potential unifying hypothesis on the underlying molecular basis of hyper-contractility caused by a majority of hypertrophic cardiomyopathy mutations
Suman Nag, Darshan V. Trivedi, Saswata S. Sarkar, Shirley Sutton, Kathleen M. Ruppel, James A. Spudich
doi: https://doi.org/10.1101/065508
Suman Nag
1Department of Biochemistry, Stanford University School of Medicine, Stanford, CA 94305.
Darshan V. Trivedi
1Department of Biochemistry, Stanford University School of Medicine, Stanford, CA 94305.
Saswata S. Sarkar
1Department of Biochemistry, Stanford University School of Medicine, Stanford, CA 94305.
Shirley Sutton
1Department of Biochemistry, Stanford University School of Medicine, Stanford, CA 94305.
Kathleen M. Ruppel
1Department of Biochemistry, Stanford University School of Medicine, Stanford, CA 94305.
2Department of Pediatrics (Cardiology), Stanford University School of Medicine, Stanford, CA 94305.
James A. Spudich
1Department of Biochemistry, Stanford University School of Medicine, Stanford, CA 94305.
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Posted July 24, 2016.
Beyond the myosin mesa: a potential unifying hypothesis on the underlying molecular basis of hyper-contractility caused by a majority of hypertrophic cardiomyopathy mutations
Suman Nag, Darshan V. Trivedi, Saswata S. Sarkar, Shirley Sutton, Kathleen M. Ruppel, James A. Spudich
bioRxiv 065508; doi: https://doi.org/10.1101/065508
Beyond the myosin mesa: a potential unifying hypothesis on the underlying molecular basis of hyper-contractility caused by a majority of hypertrophic cardiomyopathy mutations
Suman Nag, Darshan V. Trivedi, Saswata S. Sarkar, Shirley Sutton, Kathleen M. Ruppel, James A. Spudich
bioRxiv 065508; doi: https://doi.org/10.1101/065508
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