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Network analysis of genome-wide selective constraint reveals a gene network active in early fetal brain intolerant of mutation
Jinmyung Choi, Parisa Shooshtari, Kaitlin E Samocha, Mark J Daly, Chris Cotsapas
doi: https://doi.org/10.1101/017277
Jinmyung Choi
1Department of Neurology, Yale School of Medicine, New Haven CT, USA
Parisa Shooshtari
1Department of Neurology, Yale School of Medicine, New Haven CT, USA
Kaitlin E Samocha
2Analytic and Translational Genetics Unit, Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA
3Program in Medical and Population Genetics, Broad Institute of Harvard and MIT, Cambridge, Massachusetts, USA
4Stanley Center for Psychiatric Research, Broad Institute of Harvard and MIT, Cambridge, Massachusetts, USA
5Program in Genetics and Genomics, Biological and Biomedical Sciences, Harvard Medical School, Boston, Massachusetts, USA
Mark J Daly
2Analytic and Translational Genetics Unit, Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA
3Program in Medical and Population Genetics, Broad Institute of Harvard and MIT, Cambridge, Massachusetts, USA
4Stanley Center for Psychiatric Research, Broad Institute of Harvard and MIT, Cambridge, Massachusetts, USA
Chris Cotsapas
1Department of Neurology, Yale School of Medicine, New Haven CT, USA
2Analytic and Translational Genetics Unit, Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA
3Program in Medical and Population Genetics, Broad Institute of Harvard and MIT, Cambridge, Massachusetts, USA
4Stanley Center for Psychiatric Research, Broad Institute of Harvard and MIT, Cambridge, Massachusetts, USA
6Department of Genetics, Yale School of Medicine, New Haven CT, USA
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Posted March 29, 2015.
Network analysis of genome-wide selective constraint reveals a gene network active in early fetal brain intolerant of mutation
Jinmyung Choi, Parisa Shooshtari, Kaitlin E Samocha, Mark J Daly, Chris Cotsapas
bioRxiv 017277; doi: https://doi.org/10.1101/017277
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