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Modeling bi-modality improves characterization of cell cycle on gene expression in single cells
Andrew McDavid, Lucas Dennis, Patrick Danaher, Greg Finak, Michael Krouse, Alice Wang, Philippa Webster, Joseph Beechem, Raphael Gottardo
doi: https://doi.org/10.1101/002295
Andrew McDavid
aDepartment of Statistics, University of Washington, Seattle, WA 98195, USA
bVaccine and Infectious Disease Division, Fred Hutchinson Cancer Research Center, Seattle, WA 98109, USA
Lucas Dennis
cNanoString Technologies, Seattle, WA 98118, USA
Patrick Danaher
cNanoString Technologies, Seattle, WA 98118, USA
Greg Finak
bVaccine and Infectious Disease Division, Fred Hutchinson Cancer Research Center, Seattle, WA 98109, USA
Michael Krouse
cNanoString Technologies, Seattle, WA 98118, USA
Alice Wang
dBD Biosciences, San Jose CA 95131, USA
Philippa Webster
cNanoString Technologies, Seattle, WA 98118, USA
Joseph Beechem
cNanoString Technologies, Seattle, WA 98118, USA
Raphael Gottardo
aDepartment of Statistics, University of Washington, Seattle, WA 98195, USA
bVaccine and Infectious Disease Division, Fred Hutchinson Cancer Research Center, Seattle, WA 98109, USA
Article usage
Posted July 10, 2014.
Modeling bi-modality improves characterization of cell cycle on gene expression in single cells
Andrew McDavid, Lucas Dennis, Patrick Danaher, Greg Finak, Michael Krouse, Alice Wang, Philippa Webster, Joseph Beechem, Raphael Gottardo
bioRxiv 002295; doi: https://doi.org/10.1101/002295
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